lecture 19 part 1
2 Video Views·Sep 30, 2024
Peace, mercy, and blessings of God be upon you.
How are you, guys? I hope you're doing well.
This is the last lecture of the Applied course,
which is lecture number 19.
Actually, I didn't expect that we would take
this many cases in this course.
But at the end of this course, I say thank God,
finally, I mean,
now I can be reassured when I say
that if someone wants to start writing reports
and doesn't need or doesn't want to go to centers,
I expect that this course,
with the How to Report course,
will be very, very enough
to get a radiologist,
I mean,
enough and very suitable
to write correct reports
and write reports in a standardized way.
And, I mean,
the things that bothered me
when I saw one of the radiologists
writing bad reports
or that he was writing bad reports
and one of the clinicians
started to criticize him and so on.
So, this was something that bothered me.
So, I expect that these two courses,
I expect that I would have done
in terms of not being something like this
or that I didn't hear something like this.
Because, in fact,
the two courses have a very large number
of different cases
that cover a very, very large percentage
of the clinical practice.
And even, I mean,
if there is something,
of course, these things don't end.
Even if there are strange cases,
strange cases,
you already know
how you can write reports
in an organized way,
in a very good way,
and to the point, I mean,
and it is clear that it is a respectable report,
thank God.
So, thank God, first and foremost,
God willing, I will collect for you
all the reports that we took
during the two courses,
the How to Report and the Apply.
For people, of course, who attended the two courses,
they will be in one folder
that people can use
in their practical life, I mean.
For those who attended
the emergency with me,
I will also collect the emergency cases
from the applied course
in one folder, and I will send them,
God willing,
the emergency reports alone
after the course is over, God willing.
Okay, let's start,
in the name of God, the Merciful, the Compassionate.
Today's cases are honestly
some of the most enjoyable cases,
and I left it on the basis that,
thank God, we finish the course with it, I mean.
So, the first case we have
is the case of Fishbone.
Actually, this is one of the very funny cases,
and it is,
the problem is,
of course,
there is a detection, I mean, of the lesion.
The patient was coming
complaining of abdominal pain,
and it was,
as you will see,
and as most of the people
who sent me, I mean,
the examination
was almost normal,
there was no problem for us
in the examination.
Most of the people
did not see the abnormality,
but when you look at
the area of the
abdomen,
or in the area of the omentum,
I mean, the area
behind the anterior abdominal wall directly,
you will feel that this area has
some smudging of the fat blades.
This area has some smudging of the fat blades.
And when it goes down a little,
you will notice this structure.
Do you see this linear structure?
This linear,
hyperdense structure,
which is transecting
the bowel wall,
extending from the omen
to the outside.
This is abnormal.
Of course, if one does not
focus on it a little,
and does not take care of it,
he can pass it completely,
and the examination was abnormal.
To make sure that
this is not an enhancing thing,
and that it is really hyperdense,
look at it in the abdomen.
You will notice that,
if we look at the abdomen,
you will notice that this structure
is still there and hyperdense
before the anterior abdominal wall.
This means that this is a hyperdense
in non-contrast.
What is the thing that can be like this?
Of course, we have to think about
a foreign body.
And we ask the patient very well
about the things
that he may have eaten
in the past period.
Unfortunately, most people
do not keep in mind
the action
that happened.
Because, as it happened in this case,
the history may be
from three or four days.
This patient did not have a history
that he ate fish
and then he had a pain in the abdomen.
No, it does not come like this
and does not link the subject together.
This is what makes the subject
Of course, the patient's question
after seeing this
abdomen,
is very strange.
What do you see?
I do not know what I ate
or what I did not eat.
In the end,
it turned out that this patient
had eaten fish
and had a fishbone.
As you can see,
this fishbone is the
transecting of the bowel
and the abdominal vein
as you can see.
In the delayed images,
you will notice
that there is some sort of
air flow
in the peritoneum.
If we look here,
you will notice that there is a minute
tiny air flow
in the area where the smudging
is. Of course,
this bowel is not stable
so it moves.
You will notice that the fishbone
is a little further from the area
where the smudging is,
but as you can see,
the bowel moves.
This minute air flow
is abnormal.
It is probably the source of the bowel
due to the transection
of the fishbone
of this bowel.
If we also notice,
we went out a little proximal,
you will notice that there is another
similar structure
in the proximal bowel.
This is another fishbone,
but this is an intraluminal.
Another fishbone
in the proximal bowel.
It is a very simple description,
but of course,
it makes a lot of difference
to pick up abnormalities.
It is very easy to miss.
Especially, there is no clear
history of the fishbone.
Pay attention to the foreign body.
These linear things,
I show you this view
so that people may not be aware
of the foreign body
and do not know how it looks
and how it can be seen in things.
A linear hyperdense structure
is seen transecting
the wall of the distal
jejunal loops
at the region of the
suprapubic region.
at the region of the
suprapubic region.
We determine as much as we can
the bowel loops as much as we can.
Now we see this structure.
What does it do exactly?
It is associated with smudging
the fat planes around it
and tiny extraluminal air foci.
There is a similar intraluminal structure.
There is another structure
that has intraluminal
and gastric lumen.
It was in the first physics.
It was in the gastric lumen
and with time,
in the proximal jejunal loop
it moved from its place
and went to the jejunal loop.
The study has no problem at all.
We say that this is
a linear hyperdense structure.
We describe this as a
structure.
This is what we use.
Translecting the wall of
the distal jejunal loops
sorry
associated with
fat stranding
and tiny extraluminal air foci.
Then we put
the diagnosis as
a possibility of foreign body
with wall porphyration is
to be considered for clinical correlation.
and there are other similar structures
as you can see the similar structure
which is the one you can see now
that's it
the option is to open operative
or to open laparoscope
and this is the view of the fishbone
which you can see
I don't know if you can see the image
or not
but this is the view
of the fishbone
which is present
transecting the bowel
ok
this is the view of the laparoscope
and they removed it
the next case
is the foreign body
we will leave it as it is
because these are important things
after that
I have a very nice case
which is
the case of
the roe-on-wye
or the roe-on-wye complication
of course I talked more than once
about the roe-on-wye complication
and people know this story
in the
how to report lecture
I said one of the most important
roe-on-wye complication
is the internal hernia
we talked before about the roe-on-wye
and we talked about
its component
and the elementary limb
and the pylobankratic limb
and the gastric pouch
and we will talk about this story later
anyway
roe-on-wye is one of the operations
that
its purpose is
specialization
and it depends on
two things
it reduces the size of the stomach
so it reduces the capacity of the stomach
so the stomach gets full
and it depends on
there is a large part of the food
that bypasses a large part of the bowel
so there is no absorption
of large amounts of food
so it helps
specialization
roe-on-wye has two purposes
its purpose is to reduce the amount of food
due to reduction of volume of the stomach
and also to reduce the absorption
of the food by bypassing
what happens and what they do
ok
this sample was brought
from abdominal pain after roe-on-wye
and actually
when we look at the test
we will notice
the following
we will notice that
the mesenteric area
and the mesenteric fat planes
you feel that it has
small jelly fat planes
and especially
in the distal abdomen
or in the pelvic region
and the bowel loops
in the distal ileal bowel loops
their enhancement pattern
is a little less
than the other bowel loops
so
this is a little
worrying especially after roe-on-wye
and you will notice
that there are tubular structures
within the mesenteric fat planes
these tubular structures
within the fat planes
are continuous
if I show you the colonel
it will be clearer
this is the mesentery
it is very smudgy and misty
and the attenuation is very high
this is not normal
and the tubular structures
are not enhancement
this is superior mesenteric artery
next to the superior mesenteric artery
you will notice that the superior mesenteric veins
these tubular structures are not illuminated
there is
non-opacification of
the
structures
or vessels or veins
of the superior mesenteric vein
this is the artery
next to the vein
the artery is illuminated
this is the venous phase
when we come to
the origin of the superior mesenteric vein
when we look inside
we will notice that
this is the portal
and this is the splenic
and this is supposed to be the beginning
of the superior mesenteric
when we look at the superior mesenteric
this is the superior mesenteric
vein and this is the artery
we will notice that
the superior mesenteric vein
is very narrowing
and disappear
this is the superior mesenteric vein
and after that
what we see here is the superior mesenteric
artery and the veins
after that
the venous tributaries
are not illuminated
there is an occlusion
of the superior mesenteric vein
and this explains
or explains
why the bowel
which is distally
has a pore enhancement
and also
the mesenteric fat planes
the high attenuation
is the result of the occlusion
of the superior mesenteric vein
ok
let's come to a reason
what makes the superior mesenteric vein
occluded in this story
when we look at this
we will notice
that there is
some sort of
wire ball sign
do you see this
this is the
wire ball sign
look at this
you will notice that
there is a
wire ball sign
in this area
the wire ball sign
is the sign of volvulus
and the volvulus
is very easy to
occlude the vein
and also occlude the artery
and this is very well known
when the volvulus occurs
the vein and artery
are occluded
this may be the reason
for the occlusion of the vein
ok
as I said before
guys
the volvulus
in the veins of the superior mesenteric vein
is the sign of
internal hernia
as I said
the most important complications
after the superior mesenteric vein
is the internal hernia
and the internal hernia
after the superior mesenteric vein
has other signs
because of the complexity
of the process
the process of the vein
is very complex
and its anatomy changes
and the instance of the internal hernia
is very high
so they put more than one sign
if you see it
to say that there is an internal hernia
even if the typical pattern of the internal hernia
does not exist
the typical pattern of the internal hernia
is the dilated bowels
the sac configuration
the closed loop obstruction
that we know
you may not see it
in the internal hernia
the post row on y
and they relied on other signs
among the other signs
I say that this is an internal hernia
even if I do not see the typical pattern
of the internal hernia
is the volvulus
so the wire bowl sign
is a sign of volvulus
but in the post row on y
to me this is an indication of internal hernia
you will also notice
if you look here
on the third part of the dedenum
you will notice that
the third part should go through the midline
you will notice here
that there is a twist
look here on the third part of the dedenum
it should go like this
no, there is a twist
it does not go through the midline
and this other bowl loops
This is a very famous scene in the science of the lanterns here, they call it a mushroom shape masonry.
The masonry acts like a mushroom shape, as if this bowl is the stump of the mushroom, and the rest of the bowl is the head of the mushroom.
This is the appearance of the mushroom shape, and the twist is the signs of internal hernia.
This is described as internal hernia, and if you saw it in the beginning, you could say it has signs of volvulus.
I have two signs of volvulus, which for me are indirect signs of internal hernia.
The first is the mushroom shape appearance of the bowl, and the second is the wire ball appearance of the masonry.
These two signs explain to me why the masonry is closed, and explain to me why the masonry has congested masonry, and the rest of the distal loops are closed.
Some people may doubt this information, I am not sure, especially since the science is very clear here.
Some people may say that this is a result of a change of the complexity of the anatomy of the bowl, and the shape of the bowl changes in this way.
I have a clear evidence that the masonry is closed, and this is definitely pathological, but it can help me a lot if the masonry has old movies.
Before the masonry is closed, I will show you a sample of the same masonry, but before the abdominal vein.
This was in 2022, and in 2024 the masonry was closed.
When we look at the area of the superior mesenteric vein, we will notice that the location of the superior mesenteric vein is in front of the superior mesenteric artery.
Thank you for watching.

